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In this context, one in vitro study has found that glutamine causes membrane depolarization initiation and, subsequently, calcium entry into cells, which ultimately leads to GLP-1 secretion [14]

HT-29 cells were stimulated for 24 h at 37 C

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87 reported that targeted overexpression of eNOS in apolipoprotein E knockout animals accelerated rather than decreased atherosclerotic lesion formation

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